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Abdominal Distention and Constipation

Bradford P. Smith

Abdominal distention may be caused by feed, fluid, gas, feces, or a neoplasm (Boxes 7.13 and 7.14). Pregnancy or extreme obesity may also result in an enlarged abdomen.

The physical examination should determine which of these is the most likely cause. Often in ruminants the distention can be seen as primarily left sided, right sided, or bilateral. For example, bloat in ruminants results in a characteristic high left-sided gas disten­tion. It may be primary or associated with vagal indigestion, tetanus, or hypocalcemia. With vagal indigestion the rumen

■ BOX 7.11

■ BOX 7.13

Causes of Blood, Fibrin, or Mucus in Feces of the Horse

Common Causes

Foreign body

Rectal tear or trauma

Intussusception

Blister beetle (cantharidin) toxicity

Colitis, unknown cause

Salmonellosis

Less Common Causes

Purpura hemorrhagica

Small strongyle infection (cyathostomiasis) Colorectal polyps

Eosinophilic gastroenteritis

Acorn or oak poisoning

Arsenic toxicity

Organophosphate toxicity

Warfarin poisoning or other coagulation disorder Mycotoxicoses

Besnoitiosis (globidiosis) (exotic)

■ BOX 7.12

Causes of Blood, Fibrin, or Mucus in Feces of Ruminants

Common Causes

Foreign body Intussusception

Coccidiosis Salmonellosis

Less Common Causes

Rectal tear or trauma Rectal examination trauma Volvulus, root of mesentery Malignant catarrhal fever

Enterotoxemia Bovine viral diarrhea

Arsenic toxicity Abomasal torsion

Warfarin poisoning or other coagulation disorder Castor bean (Ricinus) poisoning

Tung tree (Aleurites) poisoning Solanum (nightshade, potato) poisoning Sesbania (rattlebox) poisoning

Bracken fern

becomes enlarged and fluid filled, often giving a pear shape to the abdomen as it is viewed from the rear, or a pear shape on the right and an apple shape on the left (“papple” shape) if some degree of bloat is also present.

Hypocalcemia and hypokalemia contribute to ileus and may result in constipation and abdominal enlargement. In sheep, abomasal impaction and enlargement associated with abomasal emptying defects can result in an enlarged abdomen with decreased food intake. When a mass (most commonly an abscess, a tumor, or fat necrosis [cattle only]) obstructs fecal passage, abdominal enlargement can become severe. With obstructive disease, some degree of colic is almost always present. A ruptured bladder results in a large fluid-filled abdomen, but constipation is not an obvious sign.

Causes of Abdominal Distention and Constipation in Horses

Common Causes

Ileus impaction

Intestinal foreign body such as enterolith (see Colic) Peritonitis

Intestinal obstruction, impaction, or gas (see Colic) Necrotizing enterocolitis (foals)

Torsion or volvulus of gut (see Colic) Sudden decrease in exercise

Less Common Causes

Pregnancy

Pelvic mass (abscess, tumor) Cecal tympany (see Colic) Hernia, obstructive (see Colic) Intussusception (see Colic)

Uncommon Causes

Anticholinergics

Opiates Intrinsic colonic nerve dysfunction

Anorectal pain Perineal hernia

Hypokalemia Tetanus

Hypocalcemic tetany Intramural hematomas on gut Propylene glycol toxicity Grass sickness (exotic)

The most common causes of decreased fecal output in ruminants and horses are decreased feed intake and dehydra­tion. In such cases the animal will appear gaunt or have a relatively empty abdomen or rumen. Horse feces in cases of prolonged transit are often covered with a layer of tenacious, thick, yellow mucus. When a functional obstruction (ileus, vagal indigestion) or physical obstruction (impaction, foreign body, displaced intestine, fat necrosis) occurs as a cause of constipation, the abdomen is more likely to appear normally full or to become distended. Rectal examination is of great help in determining whether a mass or an obstruction exists because loops of distended small bowel can sometimes be palpated in the latter case.

Radiographs and ultrasound may be valuable to help determine the cause of abdominal distention in foals, calves, and small ruminants. Increased gastrointestinal gas may result in abdominal distention. Abomasal bloat and necrotizing enterocolitis in young animals may best be confirmed with lateral abdominal radiographs.

Dehydration may also result in dry feces but not in abdominal enlargement. When constipation is present and feces are drier than normal, rehydration and correction of hypocalcemia, hypokalemia, and any existing acid-base abnormalities are important parts of correction of the constipation. Other nonspecific therapies for functional constipation include laxa­tives, cathartics, and cholinergic drugs. When treating constipa­tion, which is usually a secondary problem, it is important to simultaneously attempt to diagnose the primary disease.

In ruminants, when abdominal distention involves the rumen or is caused by pregnancy or obesity, colic is absent. When abdominal distention is the result of obstruction from the pylorus distal, colic is usually present. Abdominal distention and constipation are frequently accompanied by colic in the horse, regardless of anatomic site involved (review the approach to colic).

■ BOX 7.14

■ BOX 7.15

Causes of Abdominal Distention and Constipation in Ruminants

Common Causes

Pregnancy Obesity

Vagal indigestion Grain overload

Bloat

Ileus

Cecal volvulus or dilation with ileus Peritonitis, traumatic or other cause Fat necrosis involving rectum or colon Ruptured bladder (uroperitoneum) Intestinal obstruction

Pelvic mass (abscess, tumor) Hypocalcemia

Omasal obstruction or foreign body

Less Common Causes

Anticholinergics Intussusception

Abomasal volvulus Abomasal impaction

Tetanus Abomasal bloat (calf) Necrotizing enterocolitis (calf)

Uncommon Causes

Hydrops Ascites

Torsion of descending colon

Internal herniation, especially diaphragmatic hernia involving reticulum

Displacement of intestine to left of rumen Stenosis of duodenum

Adhesions of intestine Bovine leucosis

Intestinal volvulus Atresia of anus, colon, rectum, or intestine Abomasal adenocarcinoma

Omental bursitis Perforated abomasal ulcer

Zinc toxicity Crude oil toxicity

Diesel fuel toxicity Propylene glycol toxicity

Larkspur poisoning

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Source: Smith Bradford P., Van Metre David C., Pusterla Nicola (eds.). Large Animal Internal Medicine. Part 1. 6th edition. — Elsevier,2020. — 2279 p.. 2020

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