Abstract
Adiaspiromycosis, classically caused by Emmonsia species, is primarily a pulmonary disease affecting small mammals, especially members of the orders Rodentia and Carnivora. The disease name derives from the tissue form of the fungus (adiaspores), which develops when the inhaled conidia of the mycelial form of the fungus present in soil enlarge in lungs to produce thick-walled non-replicative structures.
Emmonsia crescens has apparently worldwideA. M. Borman
UK National Mycology Reference Laboratory, Public Health England, Bristol, UK e-mail: Andy.Borman@uhBristol.nhs.uk
Y. Jiang
Department of Dermatology, The Affiliated Hospital, Guizhou Medical University, Guiyang, China
Center of Expertise in Mycology Radboudumc/CWZ, Nijmegen, The Netherlands
e-mail: jiangyanping119@163.com
K. Dukik
Westerdijk Fungal Biodiversity Institute, Utrecht, The Netherlands
e-mail: k.dukik@westerdijkinstitute.nl
L. Sigler
Microfungus Collection and Herbarium [now UAMH Centre for Global Microfungal Diversity] and
Biological Sciences, University of Alberta, Edmonton, Canada
e-mail: Lynne.Sigler@ualberta.ca
I. S. Schwartz
Division of Infectious Diseases, Department of Medicine, University of Alberta, Edmonton, AB, Canada
e-mail: ilan.schwartz@ualberta.ca
G. S. de Hoog (*)
Center of Expertise in Mycology RadboudUMC/CWZ, Nijmegen, The Netherlands
Westerdijk Fungal Biodiversity Institute, Utrecht, The Netherlands
e-mail: s.hoog@westerdijkinstitute.nl
© Springer International Publishing AG, part of Springer Nature 2018 147
S. Seyedmousavi et al. (eds.), Emerging and Epizootic Fungal Infections in Animals, https://doi.org/10.1007/978-3-319-72093-7_7
distribution and is associated with very large adiaspores (up to 400 μm). The type species of Emmonsia, E. parva, however, is now recognised to be a Blastomyces species and its ‘small adiaspores' are comparable to the broad-based budding cells current in that genus. This chapter briefly summarises current knowledge concerning taxonomy, epidemiology, biology and clinical syndromes of the principal etiological agents of adiaspiromycosis in mammals.
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