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Terminology and Description of Clinical Signs of Neurologic Disease (See Table 8.1)

Telencephalon (cerebrum, basal ganglia) and diencephalon (thalamus)

Changes in behavior

Changes in the level of consciousness

Dullness, obtundation

Stupor

Coma

Excitement, mania

Seizures (convulsions)

Narcolepsy

Vision disturbance

Blindness in both visual fields (amaurosis)

Blindness in the contralateral visual field (hemianopsia) Menace reflex deficit

Change in pupil size: small to pinpoint pupils Circling (toward the side of the lesion) Head turn (toward the side of the lesion) Gait usually normal Abnormal postural reactions (contralateral)

Decreased or absent conscious proprioception

Noticeable ataxia, paresis (weakness), and paralysis are uncommon

Abnormal spinal reflexes

Normal to increased (hyperreflexic) myotatic reflexes

Altered muscle tone

Normal to increased muscle tone (spasticity)

Urinary incontinence (upper motor neuron)

Tremors

Mesencephalon (midbrain)

Changes in the level of consciousness

Dullness (depression)

Stupor

Coma

Narcolepsy

Abnormal posture

Opisthotonos

Decerebrate posture

Abnormal visual or ocular function

Blindness in both visual fields (amaurosis)

Blindness in the contralateral visual field (hemianopsia) Change in pupil size

Small pupils in early, mild lesions

Dilated, nonresponsive pupils in severe lesions

Menace reflex deficit (ipsilateral)

Anisocoria (asymmetric lesions)

Circling (toward the side of the lesion—ipsiversive) Head turn (toward the side of the lesion—ipsiversive) Abnormalities of gait (usually contralateral to lesion) Decreased or absent conscious proprioception Ataxia

Paresis (weakness)

Paralysis

bgcolor=white>Hyperphagia or hypophagia
■ TABLE 8.1
Localization of Central Nervous S ystem Lesions According to Major Signs Encountered
Sign or Problem Encountered Lesion Location
Changes in Gait and Locomotion
Ataxia Spinocerebellar tracts, spinal cord, and brainstem; vestibular system; cerebellum
Conscious proprioceptive deficit Fasciculus gracilis and fasciculus cuneatus and connections, spinal cord, brainstem, forebrain
Knuckling Usually LMN, ventral horn gray matter, spinal cord; occasionally UMN, spinal cord, brainstem
Abduction or adduction Spinocerebellar tracts, spinal cord, brainstem, cerebellum
Abnormal postural placement Spinocerebellar tracts, spinal cord, and brainstem; gracilis and fasciculus cuneatus and connections, spinal cord and brain
Hypermetria Cerebellum, cerebellar peduncles, spinocerebellar tracts
Circling, or falling to one side Forebrain, vestibular nuclei and connections, cerebellum
Paraplegia UMN, T2 to S2, or LMN, L3 to S2
Hemiplegia Ipsilateral brainstem or spinal cord, cranial to T3
Changes in Sensorium and Behavior
Coma or semicoma ARAS; diffuse forebrain
Obtundation ARAS; forebrain
Seizures Forebrain
Head pressing, compulsive walking Forebrain
Aggression or rage Forebrain
Inappropriate sexuality Forebrain
Hypothalamus
Diabetes insipidus Hypothalamus
Headshaking Unknown, probably peripheral trigeminal neuralgia
Changes in Head Posture
Stiff neck Meninges, cervical spine
Head tilt Vestibular nuclei and connections (medulla oblongata, cerebellum)
Head tremor Cerebellum, basal ganglia
Opisthotonos Cerebellum (rostral vermis), rostral brainstem, cerebrum, cranial nerve VIII
Cranial Nerve Dysfunction
Blindness Cerebral cortex, internal capsule, optic tracts, optic chiasm, optic nerve, eye
Anisocoria Cervical spinal cord (tectotegmentospinal tract), cranial thoracic nerve roots, vagosympathetic trunk, midbrain (oculomotor nerve nucleus), cranial cervical ganglion, ciliary ganglion, oculomotor nerve, optic nerve
Mydriasis Oculomotor nerve, midbrain, optic nerve, eye
Miosis Vagosympathetic trunk, ciliary ganglia, tectum, brainstem, cervical spinal cord

Facial nerve, vagosympathetic trunk, cranial cervical ganglion, midbrain, oculomotor nerve, cervical spinal cord (tectotegmentospinal tract)

Ptosis
Strabismus
Ventrolateral Cerebellum, vestibular nucleus, oculomotor nerve
Dorsomedial Trochlear nerve
Medial Abducent nerve
Nystagmus
Horizontal Nerve VIII (peripheral)
Vertical or rotatory Vestibular nuclei, peripheral vestibular receptor, cerebellum, vestibulocochlear nerve
Jaw drop Trigeminal motor nucleus (pons), trigeminal nerve
Flaccid tongue Hypoglossal nucleus (medulla oblongata), hypoglossal nerve, lingual muscle
Facial paralysis Facial nucleus (medulla oblongata), facial nerve, facial muscles
Facial analgesia Trigeminal nerve (sensory component), pons, forebrain
Dry eye Cranial nerve VII before exiting petrous temporal bone
Changes in Reflexes
Patellar L3-L6 spinal cord, femoral nerve, quadriceps femoris muscle
Flexors (thoracic limbs) C5-T2 spinal cord segments, radial, ulnar, musculocutaneous and median nerves, and innervated muscles
Flexors (pelvic limbs) L6-S2 spinal cord segments (pelvic limbs); sciatic, peroneal, and tibial nerves; flexor muscles of the limbs
Triceps C6-T1 spinal cord segments, radial nerve, triceps muscle
Cutaneous trunci C8-T1 spinal cord segments, lateral thoracic nerve, white matter of thoracic spinal cord
Anal S1-S5 sacral spinal cord segments, pudendal nerve
Cervicofacial Dorsal columns of cervical spinal cord segments; facial nucleus (medulla oblongata), nerve, facial nucleus, muscles of ear; connections between cervical plexus and facial nerve
Dysuria (dribbling urine) Spinal cord, S1-S5, sacral nerves, bladder wall

ARAS, Ascending reticular activating system; LMN, lower motor neuron; UMN, upper motor neuron.

Abnormal spinal reflexes

Normal to increased (hyperreflexic) myotatic reflexes

Altered muscle tone

Spasticity

Urinary incontinence (upper motor neuron) Metencephalon (pons, cerebellum)

Abnormal posture

Head tilt

Decerebellate posture

Circling (usually away from side of lesion—paradoxical signs) Head turn (usually away from side of lesion—paradoxical signs) Nystagmus (variable—may be constant, positional, direction changing, or disconjugate and may occur in any direction) Abnormalities of gait

Ataxia

Dysmetria—typically, hypermetria

Abnormal spinal reflexes (occasional)

Normal to increased myotatic reflexes (hyperreflexia) Altered muscle tone

Normal to increased muscle tone on the opposite side of the body (contralateral spasticity)

Normal to decreased muscle tone on the same side of the body (ipsilateral hypotonus)

Urinary incontinence (upper motor neuron) (rare) Medulla oblongata

Changes in the level of consciousness

Dullness, obtundation

Abnormal posture

Head tilt (toward side of lesion—ipsiversive)

Circling (toward side of lesion—ipsiversive)

Head turn occasionally (toward side of lesion) Strabismus—variable

Nystagmus—spontaneous, abnormal (variable—may be constant, positional, direction changing, or disconjugate and may occur in any direction)

Dysphagia

Facial analgesia

Facial paresis or paralysis

Menace reflex deficit

Jaw weakness

Roaring, snoring, dysphonia

Tongue weakness, deviation, or paralysis

Abnormalities of gait—ipsilateral

Decreased or absent conscious proprioception Ataxia

Paresis (weakness)

Paralysis

Abnormal spinal reflexes

Normal to increased (hyperreflexic) myotatic reflexes

Altered muscle tone

Normal to increased muscle tone

Urinary incontinence (upper motor neuron)

Spinal cord C1-C5

Abnormalities of gait in thoracic and pelvic limbs—ipsilateral Decreased or absent conscious proprioception Ataxia

Paresis (weakness)

Paralysis

Abnormal spinal reflexes—ipsilateral Hyperreflexia in both thoracic and pelvic limbs Decreased to absent caudal cervical and auricular reflexes Decreased to absent slap test (horses)

Altered muscle tone

Normal to increased muscle tone

Urinary incontinence (upper motor neuron)

Spinal cord C6-T2

Abnormalities of gait in thoracic and pelvic limbs—ipsilateral Decreased or absent conscious proprioception Ataxia

Paresis (weakness)

Paralysis

Abnormal spinal reflexes—ipsilateral Hyporeflexia in thoracic limbs Hyperreflexia in pelvic limbs Decreased to absent caudal cervical and auricular reflexes Decreased to absent slap test (thoracolaryngeal reflex, horses)

Absent panniculus reflex

Horner's syndrome (ipsilateral)

Altered muscle tone

Decreased muscle tone in thoracic limbs

Normal to increased muscle tone in pelvic limbs

Urinary incontinence (upper motor neuron)

Spinal cord T3-L2

Abnormalities of gait in pelvic limbs only—ipsilateral Decreased or absent conscious proprioception Ataxia

Paresis (weakness)

Paralysis

Abnormal spinal reflexes—ipsilateral Hyperreflexia in pelvic limbs only Decreased panniculus reflex caudal to lesion

Altered muscle tone—ipsilateral

Normal to increased muscle tone in pelvic limbs Urinary incontinence (upper motor neuron)

Spinal cord L3-S3

Abnormalities of gait in pelvic limbs only—ipsilateral Decreased or absent conscious proprioception Ataxia

Paresis (weakness)

Paralysis

Abnormal spinal reflexes—ipsilateral

Hyporeflexia in pelvic limbs only

Altered muscle tone

Decreased muscle tone in pelvic limbs

Flaccidity of the tail

Urinary incontinence (lower motor neuron) Fecal incontinence (lower motor neuron) Peripheral nerve and muscle

Abnormalities of gait

Paresis to paralysis

Decreased or absent conscious proprioception

Ataxia

Paresis (weakness)

Paralysis

Abnormal spinal reflexes—ipsilateral

Hyporeflexia

Altered muscle tone

Decreased muscle tone

Muscle atrophy Flaccidity of the tail

Urinary incontinence (lower motor neuron)

Fecal incontinence (lower motor neuron)

The clinical signs of neurologic disease depend on the location of the disease process within the nervous system (Table 8.1). Widely varying disease entities may produce similar or identical clinical signs.

Seizures, for example, may be the result of metabolic, toxic, traumatic, neoplastic, or other causes.
Definitive diagnosis of neurologic disease, therefore, cannot be made based on clinical signs alone. Localization of lesions within the nervous system by meticulous physical and neurologic examination is the first and key step in developing a differential diagnosis list and a rational diagnostic and therapeutic plan.

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Source: Smith Bradford P., Van Metre David C., Pusterla Nicola (eds.). Large Animal Internal Medicine. Part 1. 6th edition. — Elsevier,2020. — 2279 p.. 2020

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